BLIS M18 is a special kind of S. salivarius
Not all strains of S. salivarius act equally to inhibit
oral and dental pathogens. BLIS M18 is a naturally
occurring strain that is especially potent at inhibiting
S. mutans, the oral bacteria widely accepted to be a
main cause of dental caries and tooth decay.
BLIS M18 with Punch
In addition, M18 goes above and beyond regular
probiotic activity because it produces anti-microbial
proteins called BLIS (Bacteriocin-Like Inhibitory
Substances). Most other probiotics work by
competing with bacteria for space and nutrients in the
mouth and in this way help prevent over-growth of
bad bacteria. BLIS M18 is different. Not only does it
compete for space and nutrients but it also produces
three potent antibacterial BLIS molecules; Salivaricin A, Salivaricin 9, Salivaricin X
A Unique Enzyme
Finally, M18 has unique enzyme producing abilities
that allow it to break up dental plaque and neutralize
acid that can be harmful to teeth and gums. This is
a unique attribute of BLIS M18 and not found in any
other oral probiotics. As a result of its multi-functional activity, taking daily
doses of BLIS M18 can Protect teeth from bacteria
known to cause tooth decay,
Protect teeth from acid erosion
caused by high acid foods, and
help break up dental plaque.
Supported by Proprietary Science
BLIS M18 Protects from dental pathogens
Streptococcus mutan is considered a leading
causative agent of tooth decay. In vitro studies have
shown BLIS M18 is a potent inhibitor of this bacteria.
BLIS M18 Protects teeth from acid erosion
Organisms responsible for dental plaque like
S. mutans produce lactic acid from dietary
carbohydrate. Lactic acid degrades tooth enamel and
dentine (a major component of teeth) which can
lead to cavities and eventually tooth loss. Research
has shown that M18 produces an enzyme called
urease which neutralizes acid in the oral cavity.
BLIS M18 Breaks up dental plaque
While Streptococcus mutans are able to produce acid
they also produce extracellular polysaccharides which
contributes to the plaque biofilm or the sticky structure
which allows the problematic bacteria to be in close
association with dental tissue. Limited numbers of
S. salivarius strains, including M18, produce an
enzyme called dextranase, which can help break up
the plaque biofilm caused by S. mutans. |